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Tips for care of Slow or Irritable Bowel (IBS) Ulcerative Colitis or Spastic Colon There are many factors to take into account in considering tips for the care of a slow or irritable bowel. 1. Functional Neurology Check-up
As research has shown that the cause of IBS is related to neuro-endocrine immune system dysfunction (brain and stomach hormones) – it is suggested that a functional neurologist may be able to assist. A functional neurologist, can do a proper neuro examination, determine level of neuro dysfunction and if appropriate prescribe specific non-invasive exercises to help improve the neuro-endocrine immune system function. Functional Neurology is a relatively new area pioneered by Dr. Carrick of the Carrick Institute, USA. 2. Nutritional Therapy - Consult your nutritional therapist or other health professional with appropriate professional knowledge in this area. Drs. at GFC have a special interest in nutrition.
- Correction of poor eating habits, this usually involves increasing fibre, reducing coffee, junk food and simple sugars.
- Avoid any known allergens including gluten for Celiac disease, common allergens include wheat, yeast, dairy, peanuts, chemicals and some preservatives and some colouring agents etc.
- Boost optimal stress coping chemicals with intake of magnesium, B-vitamins and serotonin precursor such as 5-Hydroxy-Tryptophan.
- Restore normal gut flora with probiotics.
- Consider use of digestive enzymes with meals.
- Some Herbs may be useful in restoring digestive balance.
- Fibre supplements may be useful.
3. Reduce Physical and Emotional Stress Changes or alteration in the alignment of the digestive tract or of surrounding skeletal structures for example the spine and pelvis can produce functional problems like irritable bowel syndrome (IBS). Areas most often involved include the mid and lower back, pelvic misalignment as well as malposition of the digestive tract with respect to surrounding organs. A chiropractor can assist you with restoration of structural balance. 4. Reduce Bowel Toxicity The bowel is one of the major routes for disposal of waste and toxins. If its function is reduced toxicity may result from accumulating waste substances. Return of the bowel to normal function is therefore important and you should see your health professional. 5. Enhance Immune System The immune system is compromised in people with IBS. Given the vital protective role of the immune system, strengthening it will benefit the affected individual. 6. Check for Malfunction of nerves or spinal trauma impacting bowel Evidence supports that certain cases of bowel disease, such as Irritable Bowel Syndrome, result from the malfunction of nerves that control the gastrointestinal system (autonomic nervous system and/or enteric nervous system) and/or the immune system, or following trauma to the spine and/or spinal cord – more info below with references. Signs and Symptoms of Irritable Bowel Syndrome? - Cramping pain in your lower abdomen
- Relief of pain after bowel movements
- Bloating and gas
- Changes in your bowel habits
- Feeling of incomplete emptying after bowel movements
- Diarrhoea or constipation, or both alternately
- Immediate need to move your bowels when you wake up or during or after meals
- Mucus in your stool
References: 1.Bodemar G, Ragnarsson G. [Irritable bowel syndrome. Survey of definitions, differential diagnosis and pathogenesis]. Lakartidningen. 2001 Feb 14;98(7):666–71. 2.FDA Announces Discontinued Marketing of GI Drug, Zelnorm, for Safety Reasons,” published on 3/30/07 via the U.S. Food and Drug Administration Web site (www.fda.gov). Accessed 3/30/07. 3.“FDA Public Health Advisory, Tegaserod maleate (marketed as Zelnorm)” published on 3/30/07 via the U.S. Food and Drug Administration Web site (www.fda.gov). Accessed 4/3/07. 4.“Novartis Stops Zelnorm Marketing, Sales on Heart Risk,” by Eva von Schaper. Published 4/2/07 via Bloomberg.com (www.bloomberg.com). Accessed 4/2/07. 5.Sandler RS. Epidemiology of irritable bowel syndrome in the United States. Gastroenterology. 1990;99:409-415. 6.Thompson WG, Longstreth G, Drossman DA. Functional bowel disorders and Functional abdominal pain. In: Drossman DA, Corazziari E, Talley N, Thompson WG, Whitehead W, eds. Rome II: The Functional Gastrointestinal Disorders. 2nd ed. McLean, VA: Degnon Associates, 2000:351-375. 7.Goldberg PA, “Modification ofvisceral sensitivity and pain in irritable bowel syndrome by 5-HT3 antagonism (ondansetron).” Digestion Nov-Dec 1996:57(6):478-83. 8.Saggioro A. Probiotics in the treatment of irritable bowel syndrome. J Clin Gastroenterol. 2004 Jul;38(6 Suppl):S104–S106. 9. Monthly Prescribing Reference. Prescribing Reference Inc. New York, NY. October 2006. How IUCCA Upper Cervical Care Relates to Irritable Bowel Syndrome / Ulcerative Colitis Evidence supports that certain cases of bowel disease, such as Irritable Bowel Syndrome, result from the malfunction of nerves that control the gastrointestinal system (autonomic nervous system and/or enteric nervous system) and/or the immune system, or following trauma to the spine and/or spinal cord.(ref. 1-15) In each of these cases, it is possible the upper cervical spine is involved since injury to the cervical spine can create malfunction within the autonomic nervous system, the immune system, and the nerves traveling from the brain to the gastrointestinal system.(ref. 16-32) The purpose of IUCCA upper cervical care is to reverse the trauma-induced neck injury; thereby reducing irritation to the injured nerves that supply the immune system and small and large bowel. While many bowel disease sufferers recall specific traumas such as head injuries, auto accidents or falls that preceded the onset of their symptoms, some do not. In certain pediatric cases, the injury can occur from the normal birthing process. An upper cervical examination with appropriate Radiography and Imaging is necessary in each individual's case to assess whether an upper cervical injury is present and whether benefit from upper cervical care can be achieved. References: 1. Heitkemper M, Burr RL, Jarrett M. Evidence for autonomic nervous system imbalance in women with irritable bowel syndrome. Dig Sci 1998 Sep; 43(9): 2093-8. 2. Tomita R, Munakata K, Tanjoh K. Role of non-adrenergic non-cholinergic inhibitory nerves in the colon of patients with ulcerative colitis. J Gastroenterol 1998 Feb; 33(1): 48-52. 3. Vanner S, Surprenant A. Neural reflexes controlling intestinal microcirculation. Am J Physiol 1996 Aug; 271(2 Pt 1): G223-30. 4. Knowles CH, Scott SM, Lunniss PJ. Slow transit constipation: a disorder of pelvic autonomic nerves? Dig Dis Sci 2001 Feb; 46(2): 389-401. 5. Roberts PJ, Morgan K, Miller R. Neuronal COX-2 expression in human myenteric plexus in active inflammatory bowel disease. Gut 2001 Apr; 48(4): 468-72. 6. Elsenbruch S, Orr WC. Diarrhea- and constipation-predominant IBS patients differ in postprandial autonomic and cortisol responses. Am J Gastroenterol 2001 Feb; 96(2): 460-6. 7. Sharkey KA, Kroese AB. Consequences of intestinal inflammation on the enteric nervous system: neuronal activation induced by inflammatory mediators. Anat Rec 2001 Jan 1; 262 (1): 79-90. 8. Kobayashi H, Hirakawa H, Puri P. Is intestinal neuronal dysplasia a disorder of the neuromuscular junction? J Pediatr Surg 1996 Apr; 31 (4): 575-9. 9. Lembo T, Munakata J, Mertz H. Evidence for the hypersensitivity of lumbar splanchnic afferents in irritable bowel syndrome. Gastroenterology 1994 Dec; 107(6): 1686-96. 10. Sanovic S, Lamb DP, Blennerhassett MG. Damage to the enteric nervous system in experimental colitis. Am J Pathol 1999 Oct; 155(4): 1051-7. 11. Ammann K, Stoss F, Meier-Ruge W. Intestinal neuronal dysplasia in adults as a cause of chronic constipation: norphometric characterization of colon innervation. Chirurg 1999 Jul; 70(7): 771-6. 12. Khurana RK, Schuster MM. Autonomic dysfunction in chronic intestinal pseudo-obstruction. Clin Auton Res 1998 Dec; 8(6):335-40. 13. Harrison JR, Blackstone MO, Vargish T. Chronic intermittent intestinal obstruction from a seat belt injury. South Med J 2001 May; 94(5): 499-501. 14. Lynch AC, Wong C, Anthony A. Bowel Dysfunction following spinal cord injury: a description of bowel function in a spinal cord-injured population and comparison with age and gender matched controls. Spinal Cord 2000 Dec; 38(12): 717-23. 15. Krogh K, Mosdal C, Laurberg S. Gastrointestinal and segmental colonic transit times in patients with acute and chronic spinal cord lesions. Spinal Cord 2000 Oct; 38(10):615-21. 16. Sato A. The somatosympathetic reflexes: their physiologic and clinical significance. In: Goldstein M, ed. The research status of spinal manipulative therapy. Washington, DC: Government Printing Office. 1975:163-172. 17. Kiyomi K. Autonomic system reactions caused by the excitation of somatic afferents: study of cutaneo-intestinal reflex. In: Korr IM, ed. The neurobiologic mechanisms in manipulative therapy. New York: Plenum, 1978:219-227. 18. Klougart N, Nilsson N, Jacobsen J. Infantile colic treated by chiropractors: a prospective study of 316 cases. JMPT 1989;21:281-288. 19. Nilsson N. Infantile colic and chiropractic. Eur Jour Chiro 1985;33:264-265. 20. Coote, J. Somatic Sources of Afferent Input as Factors in Aberrant Autonomic, Sensory, and Motor Function. In: Korr, I., ed. The Neurobiologic Mechanisms in Manipulative Therapy. New York: Plenum, 1978:91-127. 21. Denslow, J., Korr, I., Krems, A. Quantitative Studies of Chronic Facilitation in Human Motorneuron Pools. Am J Physiol 1987;150:229-238 22. Korr, I. Proprioceptors and the Behavior of Lesioned Segments. In: Stark, E. ed. Osteopathic Medicine. Acton, Mass.: Publication Sciences Group, 1975:183-199. 23. Sato, A. The somatosympathetic reflexes: their physiological and clinical significance. In: Golstein M, ed. The research status of Spinal Manipulative Therapy. Washington D.C.: Government Printing Office 1975: 163-172. 24. Sato A, Schmidt RF. Somatosympatheitc reflexes: afferent fibers, central pathways, discharge characteristics. Phys Review 1973; 53:916-947. 25. Kiyomi K. Autonomic system reactions caused by excitation of somatic afferents: study of cutaneo-intestinal reflex. In: Korr IM, ed. The neurobiological mechanisms in manipulative therapy. New York: Plenum 1978:219-227. 26. Wick, G., et al. Immunoendocrine Communication via The Hypothalamus-Pituitary-Adrenal Axis in Autoimmune Diseases. Endocrine Reviews. 14:539-563, October 1993. 27. Black, P. Immune System - Central Nervous System Interactions: Effect and Immunomodulatory Consequences of Immune System Mediators on The Brain. Antimicrobial Agents and Chemotherapy. 38:7-12, January 1994. 28. Ader, R., Cohen, N., Felten, D. Psychoneuroimmunology: Interactions Between The Nervous System and The Immune System. Lancet 345:99-103, January 14, 1996. 29. Denckla WD. Interactions between age and the neuroendocrine and immune systems. Fed Proc 1978;37:1263-1267 30. Van Dijk H, Jacobse-Geels H. Evidence for the involvement of corticosterone in the ontology of the cellular immune apparatus of the mouse. Immunology 1978;35:637-642 31. Settipane GA, Pudupakkam RK, McGowan JH. Corticosteroid effect on immunoglobins. J Allergy Clin Immunol 1978;62:162-166. 32. Korr IM. Sustained sympathecotonia as a factor in disease. In:Korr IM, ed. The neurobiological mechanisms in manipulative theraphy. New York: Plenium, 1978 229-268. 33. Roger Murphree, D.C. Alabama, USA. Article on IBS
34. Dr. Sean Holmes, medical Dr. and Chiropractor, Total Health Care.
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